Last time | Next time |
My main observation is that, by looking at others' work, you have identified ways in which you can improve your own. That's a very positive thing!
If you need any help, please come and see me. Feel free to email with questions, as well.
A couple of things to point out (here's my annotated pdf):
The "PDO" -- The Pacific Decadal Oscillation -- is a well-known source of long time-scale variability:
There is also an AMO (guess what that means?:). It might be more relevant for Togo:
Nathan felt that the 24 year periodic sine his group fit to the Niamtougou data might have been "ridiculous", but there's nothing ridiculous about it at all! There are big things out there lurking, some of which we are unaware of.
Adam observed something that I did, too: the Sotouboua minima seems to have a "U or V shaped curve every twenty years or so, which could possibly be modeled with a periodic function."
So Nathan, we don't think that you're being ridiculous!:)
My young friends, in all honesty, this is a truly conservative lower estimate: sea level is likely to far exceed this amount.
My personal experience in this area of climate change research is that "worst case scenarios" have been exceeded over and over. That's not supposed to happen, but it illustrates the non-linear nature of the phenomena involved, and our lack of understanding of the interconnectedness of nature.
Our National Intelligence agencies are worried (as they should be): Intelligence Agencies Warn of Climate Risks in Worldwide Threat Assessment: While top Trump administration officials deny climate change, the intelligence agencies warn global warming can fuel disasters and violent conflicts.
Here's the Mathematica file.
One question you may have is how to interpret the parameters?
where all parameters should be positive. $a$ is the asymptote, $p$ was designed to control how flat the curve starts out, and $b$ was intended to be related to growth rate of the curve, inflection, etc.
As it turned out, $b$ and $p$ are a little confounded.
We've been mostly dealing with empirical models -- we notice a relationship between two variables, and we construct a model of one as a function of the other -- without any belief, necessarily that one is causative. We study correlation, rather than causation.
Exceptions include that sea anemone data we looked at: response duration could be considered related to the square root of area, because the greater the area, the longer it takes a signal to travel out from the center; or the periodic oscillations on the Keeling data -- those sinusoidal oscillations we understand as the "breathing of the Earth's forests".
But now we want to do some modeling where we build in the relationships between the variables, like we're building some structure in your back yard. These are structural models.
I asked you to read a short introduction to modeling by some Canadian math/biology types: pages 3-8 (the 7th-12th pages of the pdf) of this intro to modeling, by De Vries, et al. (which I've marked up).
It gives us a tiny introduction to differential equations, difference equations, growth, and SIR infection models -- things we'll be studying in the week(s) ahead.
Let's take a look at that. In particular, we want to look at page 5, which outlines two different ways of thinking about modeling infectivity.